Models for Risk Assessment of Reactive Chemicals in Aquatic Toxicology Modellen voor de risico-evaluatie van reactieve stoffen in de aquatische toxicologie

نویسنده

  • Andreas Peter Freidig
چکیده

sFreidig A. P., Hermens J. L. M. (1995). Estimating the bioavailable aqueous concentra-tion of organic micropollutants with an equilibrium solid phase extraction. Second SETACWorld Congress, Vancouver, Canada.Freidig A., Verhaar H., Hermens J. (1997). A cell-physiological model for the acute toxic-ity of acrylates and methacrylates in fish, based on chemical reactivity. 18th SETAC meeting,San Francisco, USA.Freidig A. P., Verhaar H. J. M., Hermens J. L. M. (1998). Quantitative structure propertyrelations (QSPR’s) for the chemical reactivity of acrylates and methacrylates. European Sym-posium on Quantitative Structure-Activity Relationships, Copenhagen, Denmark.Freidig A., Hofhuis M., van Holstijn I., Hermens J. (1999). Glutathione depletion in pri-mary rat hepatocytes as an additive effect of different toxic mechanisms. 38th SOT meeting,New Orleans, USA.Freidig A., Hermens J. (1999). Narcosis and michael addition: comparing two differentmodes of action in aquatic toxicology. 9th SETAC-Europe meeting, Leipzig, Germany.Freidig A. P., Hermens J. L. M. (2000). Acute fish toxicity of phosphorothionate-estersdue to two independent toxic effects on the nervous system. 39th SOT meeting, Philadelphia,USA.

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تاریخ انتشار 2000